By Michel E. Safar, Edward D. Frohlich

ISBN-10: 3805581769

ISBN-13: 9783805581769

This ebook offers the present deliberating overseas specialists concerning the underlying mechanisms of cardiovascular probability in addition to the pathogenesis and pathophysiology of impaired huge arterial stiffness and decreased huge arterial distensibility. It additionally demonstrates the explanation for the current method of the administration and therapy of hypertensive ailments and atherosclerosis. The precise readership comprises uncomplicated scientists in vascular body structure in addition to physicians and scientists in cardiovascular medication, diabetes mellitus and renal ailments.

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Sample text

However, increasing aortic diameter with advancing age may also be viewed as a maladaptive attempt by the aorta to limit the age-related increase in aortic impedance that would otherwise accompany age-related stiffening of the aortic wall. Age-related arterial dilation tends to reduce (not increase) arterial impedance, thereby reducing systolic BP and PP [39]. The idea that systolic hypertension can be caused by a disproportionately small aortic diameter runs counter to older thinking and has been attacked on the basis of studies showing increased PWV, elastic modulus, and aortic wall thickness in convenience samples of older people [40–42].

The determinant of wide pulse pressure is increased aortic impedance, either due to a smaller aortic diameter or increased effective wall stiffness. At older ages, high aortic impedance is usually associated with wide pulse pressure due to increased aortic stiffness, which acts to reduce diastolic BP even when SVR is moderately elevated (see text). consequence of longstanding diastolic hypertension, as once thought. Systolic hypertension arises de novo at any age, and often preceding diastolic hypertension [6].

J Hypertens 1937;22:1937–1944. Lemogoum D, Flores G, Van den Abeele W, Ciarka A, Leeman M, Degaute JP, van de Borne P, Van Bortel L: Validity of pulse pressure and augmentation index as surrogate measures of arterial stiffness during ␤-adrenergic stimulation. J Hypertens 2004;22:511–517. Izzo /Mitchell 32 53 54 55 56 57 58 59 60 61 62 63 64 65 66 67 68 69 70 71 72 Wilkinson IB, MacCallum H, Hupperetz PC, van Thoor CJ, Cockcroft JR, Webb DJ: Changes in the derived central pressure waveform and pulse pressure in response to angiotensin II and noradrenaline in man.

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Atherosclerosis, large arteries and cardiovascular risk by Michel E. Safar, Edward D. Frohlich


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